How it works (big picture)
GLOW layers three peptides that each nudge the body’s built-in repair programs from different angles—blood-vessel growth, cell movement, and matrix remodeling—so the overall signal is broader than any single piece alone.

BPC-157:
A gastric pentadecapeptide that’s been shown in preclinical work to:

• Speed up new vessel formation (VEGFR2-linked)

• Encourage fibroblast migration and epithelial closure (FAK–paxillin pathways)

• Tune nitric-oxide signaling, which helps local blood flow and tissue repair
  Net effect in models: more organized tendon, muscle, and gut lining repair signals.

TB-500 (Thymosin β-4):
A 43-aa peptide that manages actin dynamics, the scaffolding cells use to move. Studies show:

• Faster cell migration to the injury site

• Upregulated VEGF and angiogenesis

• Broad anti-inflammatory actions and progenitor-cell mobilization
  Observed across myocardium, dermis, and connective-tissue models.

GHK-Cu:
A copper-binding tripeptide (glycyl-L-histidyl-L-lysine) noted for:

• Stimulating collagen and ECM production

• Tuning gene expression tied to remodeling and hair biology

• Antioxidant/anti-inflammatory balance via TGF-β and MMP regulation

Why they play well together (synergy)

• Angiogenesis: BPC-157 and TB-500 both lift VEGF-driven vessel sprouting; GHK-Cu supports endothelial proliferation—more routes for nutrients and cleanup.

• Cell migration + matrix work: TB-500 improves actin polymerization (cells move better); BPC-157 and GHK-Cu cue ECM synthesis and fibroblast activity—so arriving cells have scaffolding to build with.

• Inflammation & oxidative stress: All three dial down cytokine noise and oxidative stress signals, helping keep the repair environment on task rather than inflamed.

Where this matters (research context)
Taken together, the blend provides a multi-pathway repair signal seen in preclinical musculoskeletal, dermal/skin, and barrier-tissue models—useful when studying complex or slow-to-resolve tissue challenges.